Call for Papers: Special issue - Tinnitus, Hyperacusis, and Hidden Hearing Loss from Cochlea to Brain
• 大类 : 医学 - 3区
• 小类 : 神经科学 - 3区
Sensory hair cells in the cochlea transduce environmental sounds into high-fidelity neural signals that are conveyed by auditory nerve fibers to structures in the central auditory pathway. Over the past decade, it has become clear that subtle damage to hair cells or to afferent synapses on auditory nerve fibers can escape detection by routine clinical hearing assessment. Such "hidden hearing loss" may be involved in several disorders of central auditory processing including tinnitus, loudness intolerance, painful hearing, or more subtle problems such as impaired listening in noisy environments, which can appear even when the clinical audiogram is normal. When input from the cochlea to central auditory pathways is reduced or lost, forms of neural plasticity are activated that may compensate for the missing input, but at some cost. Weak cochlear signals are progressively amplified along the auditory pathway yielding larger responses than normal (increased central gain), altered tuning bandwidths, and increased spontaneous and synchronous neural activity, which may contribute in varying degrees to tinnitus, hyperacusis, and difficulties in normal listening. The goal of this Special Issue is to summarize the most recent electrophysiological, neuroanatomical, psychophysical and imaging results in humans and animal models, to gain insight into how hearing loss (hidden or explicit) leads to disorders of hearing and into how the disorders might best be treated.
If you would like to submit your article to be considered for publication in this special issue please do so at https://ees.elsevier.com/nsc and the select the article type “SI: Tinnitus Hyperacusis” from the drop down menu.